Abstract

Primary (or chronic) open-angle glaucoma (POAG or COAG) may be asymptomatic but causes progressive optic nerve damage with significant loss of visual field.^1–3 Treatments aim to lower intraocular pressure (IOP) by reducing the production of aqueous humour and/or increasing its drainage.^4,5 Here we update our previous articles^1,4 to reflect new drugs, preservative-free preparations and fixed-dose drug combinations for POAG.